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타깃 · 모달리티 · 임상 근거 · 비임상 맥락으로 최대 5개 프로그램을 나란히 비교합니다. · 다음 갱신 D-6 · 마지막 9월 2일

현재 선택: 5 · Data Tier에서 열림

프로그램 상세에서 관심 등록 후 2개 이상 모으면 여기서 한 번에 비교할 수 있습니다.

API CSV32행 · 5개 프로그램

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항목
AntibodyCurated CoreFDAApproved
adalimumab (Humira, Hyrimoz, Amjevita)
AbbVie·TNF-α
574 trials·t½ ~14–19 days (SC)
AntibodyCurated CoreFDAApproved
ustekinumab (Stelara, Wezlana)
Johnson & Johnson·IL-12 / IL-23 (p40)
195 trials·t½ 19 h
AntibodyCurated CoreFDAApproved
risankizumab-rzaa (Skyrizi, BI 655066, risankizumab)
AbbVie·IL-23 p19
110 trials·t½ 28 h
AntibodyCurated CoreFDAApproved
guselkumab (Tremfya, CNTO 1959)
Johnson & Johnson·IL-23 p19
87 trials·t½ ~15–18 days
AntibodyCurated CoreApproved
Remicade® (Remicade®)
Janssen·Ankylosing Spondylitis
406 trials
Overview
Program
Humira (adalimumab)Stelara (ustekinumab)Skyrizi (risankizumab-rzaa)Tremfya (guselkumab)Remicade®
Overview
Company
AbbVieJohnson & JohnsonAbbVieJohnson & JohnsonJanssen
Overview
Modality
ANTIBODYANTIBODYANTIBODYANTIBODYANTIBODY
Overview
Target
TNF-αIL-12 / IL-23 (p40)IL-23 p19IL-23 p19Ankylosing Spondylitis
Overview
Indication
Rheumatoid arthritis, psoriasis, Crohn's disease, ulcerative colitis, axial SpAPsoriasis, psoriatic arthritis, Crohn's disease, ulcerative colitisPlaque psoriasis, PsA, Crohn's disease, ulcerative colitisPlaque psoriasis, psoriatic arthritis, ulcerative colitis, Crohn's diseaseCrohn's Disease
Overview
Phase
APPROVEDAPPROVEDAPPROVEDAPPROVEDAPPROVED
Overview
Status
APPROVEDAPPROVEDAPPROVEDAPPROVEDACTIVE
Overview
Content status
Curated CoreCurated CoreCurated CoreCurated CoreCurated Core
Overview
Data Confidence
Data Confidence · HighData Confidence · HighData Confidence · HighData Confidence · HighData Confidence · High
Overview
Development Signal
Development Signal · EstablishedDevelopment Signal · EstablishedDevelopment Signal · EstablishedDevelopment Signal · EstablishedDevelopment Signal · Established
Overview
Approval status
FDA approvedFDA approvedFDA approvedFDA approvedApproved (flag incomplete)
Positioning
Key differentiator
novel object recognition testNovel therapeutic avenues, including TYK2 and HDAC inhibitors, as well as nanotechnology-based delivery systems, show encouraNovel therapeutic avenues, including TYK2 and HDAC inhibitors, as well as nanotechnology-based delivery systems, show encouraLeaving IL-12 intact preserves Th1 antimicrobial immunity while shutting down the Th17 axis, which is why durability is high and infection signals are low.novel alternative targets with improved efficacy and safety profiles to treat RA
Positioning
Known limitation
Anti-drug antibodies, TNF-independent inflammation, and immunogenicity lowering trough levels.IL-23 axis escape, anti-drug antibodies, and non-IL-12/23 inflammation.IL-17 independent disease and immunogenicity.Anti-drug antibodies are rare; non-IL-23-driven fibrostenotic disease does not respond.
Positioning
Development positioning
IL-23 class benchmark alongside Skyrizi, now with the broadest IBD label progression.
MoA
Mechanism
Adalimumab binds specifically to TNF-α and blocks its interaction with p55 and p75 cell-surface TNF receptors, neutralizing TNF-mediated inflammatory cascades (IL-6, adhesion molecules, acute-phase proteins).Ustekinumab binds the p40 subunit shared by IL-12 and IL-23, preventing their interaction with IL-12Rβ1 receptor and downstream Th1/Th17 differentiation and cytokine production.Risankizumab selectively binds IL-23 p19 subunit, blocking IL-23 interaction with IL-23R and downstream Th17 pathway (IL-17A/F, IL-22).Neutralizes IL-23 by binding p19, blocking IL-23R signaling and downstream Th17/Th22 differentiation, IL-17 production, and tissue inflammation in skin, joints, and gut mucosa.targeted metabolomics, to delineate microbial and metabolic signatures predictive of IFX induction efficacy and to explore mechanistic pathway
MoA
Biomarker
CRP, ESR, disease-specific scores (DAS28, PASI, Mayo for IBD).biomarkers but require external validation in independent cohortsPASI, sPGA, IBD endoscopic scores.PASI/IGA in psoriasis, ACR20 in PsA, endoscopic improvement and fecal calprotectin in IBD.biomarkers and safety profile were noted before and after switch
PK/PD
Half-life
~14–19 days (SC)19 h28 h~15–18 days
PK/PD
Species
Rat, xenografts, which corroborat, CRP reduction, clinical remission scoresMouse, Skin clearance (PASI75/90), endoscopic response in IBDMouseCynomolgus monkey, PASI 90/100, ACR20/50, endoscopic remission, serum IL-17A/IL-22Mouse, Human
PK/PD
Animal (cat.)
RatMouseMouseHuman, NHPHuman, Mouse
PK/PD
Experiment
pdPharmacokineticPharmacokineticPharmacokineticpharmacokinetic
Toxicology
Species
Cynomolgus monkey, Rat, xenografts, which corroboratCynomolgus monkey, MouseCynomolgus monkey, MouseMouse, PigMouse, Human
Toxicology
Animal (cat.)
NHPHuman, Mouse
Toxicology
Major finding
Serious infections (TB reactivation, invasive fungal), malignancy risk signal, injection-site reactions, and rare demyelinating events.Infections, reversible posterior leukoencephalopathy syndrome (rare), hypersensitivity.Upper respiratory infections, headache, fatigue; low TB reactivation vs TNF class.Upper respiratory infection, headache, and injection-site reactions dominate. Serious infection and malignancy signals have stayed low across long-term psoriasis follow-up.Multiple switches between infliximab originator and biosimilars in pediatric inflammatory bowel disease.. Infliximab (IFX) is highly effective but costly, driving the adoption of biosimilars. Data on switching from originator IFX to biosimilars in pediatric inflammatory bowel disease (IBD) patients remain limited. This study evaluated the safety and efficacy of switching from originator IFX to IFX…
Toxicology
CRS
N/AN/AN/AN/A
Clinical
Safety signal
Serious infections (TB reactivation, invasive fungal), malignancy risk signal, injection-site reactions, and rare demyelinating events.Infections, reversible posterior leukoencephalopathy syndrome (rare), hypersensitivity.Upper respiratory infections, headache, fatigue; low TB reactivation vs TNF class.Upper respiratory infection, headache, and injection-site reactions dominate. Serious infection and malignancy signals have stayed low across long-term psoriasis follow-up.Multiple switches between infliximab originator and biosimilars in pediatric inflammatory bowel disease.. Infliximab (IFX) is highly effective but costly, driving the adoption of biosimilars. Data on switching from originator IFX to biosimilars in pediatric inflammatory bowel disease (IBD) patients remain limited. This study evaluated the safety and efficacy of switching from originator IFX to IFX…
Clinical
Selected reported efficacy
PASI75 67%
Clinical
PASI75
67%
Clinical
Result source
ClinicalTrials.gov NCT02405780PHOENIXClinicalTrials.gov NCT02990806
Clinical
Program phase
APPROVEDAPPROVEDAPPROVEDAPPROVEDAPPROVED
Clinical
Trial activity
No active/completed countsNo active/completed countsNo active/completed countsNo active/completed countsNo active/completed counts
Clinical
Trial ref
NCT02405780PHOENIXNCT02990806
Preclinical
Animal (cat.)
MouseMouse