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타깃 · 모달리티 · 임상 근거 · 비임상 맥락으로 최대 5개 프로그램을 나란히 비교합니다. · 다음 갱신 D-6 · 마지막 9월 2일

현재 선택: 5 · 임상 갱신 필요 1 · Data Tier에서 열림

프로그램 상세에서 관심 등록 후 2개 이상 모으면 여기서 한 번에 비교할 수 있습니다.

API CSV34행 · 5개 프로그램

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항목
AntibodyCurated CoreFDAApproved
trastuzumab (Herceptin, Ogivri, Herzuma)
Roche / Genentech·HER2
1100 trials·t½ 4 h
AntibodyCurated CoreFDAApproved
zanidatamab (Ziihera, ZW25, zanidatamab-hrii)
Jazz Pharmaceuticals / Zymeworks·HER2 (biparatopic)
35 trials·t½ ~7 days
AntibodyCurated CoreFDAApproved
tislelizumab (Tevimbra, BGB-A317, tislelizumab-jsgr)
BeOne Medicines·PD-1
213 trials·t½ ~20 days class range
AntibodyCurated CoreFDAApproved
zolbetuximab (Vyloy, IMAB362, zolbetuximab-clzb, claudiximab)
Astellas·CLDN18.2 (claudin-18.2)
26 trials·t½ ~11 days
AntibodyCurated CorestalePhase 3
Obinutuzumab (Obinutuzumab)
Roche / Genentech·B-cell Lymphoma
153 trials
Overview
Program
Herceptin (trastuzumab)Ziihera (zanidatamab)Tevimbra (tislelizumab)Vyloy (zolbetuximab)Obinutuzumab
Overview
Company
Roche / GenentechJazz Pharmaceuticals / ZymeworksBeOne MedicinesAstellasRoche / Genentech
Overview
Modality
ANTIBODYANTIBODYANTIBODYANTIBODYANTIBODY
Overview
Target
HER2HER2 (biparatopic)PD-1CLDN18.2 (claudin-18.2)B-cell Lymphoma
Overview
Indication
HER2+ breast cancer, HER2+ gastric/GEJ adenocarcinomaFirst-line HER2-positive gastric, GEJ, or esophageal adenocarcinoma; previously treated HER2-positive biliary tract cancerEsophageal squamous cell carcinoma, gastric/GEJ adenocarcinoma, and first-line HER2-positive GEA with zanidatamab and chemotherapyCLDN18.2-positive HER2-negative advanced gastric or gastroesophageal junction adenocarcinomaBreast Adenocarcinoma; Metastatic Breast Carcinoma
Overview
Phase
APPROVEDAPPROVEDAPPROVEDAPPROVEDPHASE_3
Overview
Status
APPROVEDAPPROVEDAPPROVEDAPPROVEDRECRUITING
Overview
Content status
Curated CoreCurated CoreCurated CoreCurated CoreCurated Core
Overview
Data Confidence
Data Confidence · HighData Confidence · HighData Confidence · HighData Confidence · HighData Confidence · High
Overview
Development Signal
Development Signal · EstablishedDevelopment Signal · EstablishedDevelopment Signal · EstablishedDevelopment Signal · EstablishedDevelopment Signal · Emerging
Overview
Approval status
FDA approvedFDA approvedFDA approvedFDA approvedInvestigational
Positioning
Key differentiator
novel bifunctional chelator (2E-C-NETA) for applications in 177 Lu-based TRTDual-epitope binding drives receptor clustering, faster internalization, and stronger signaling blockade than either monoclonal alone. In GEA the label splits IHC 3+ (chemo doublet) from IHC 3+ or 2+/ISH+ (chemo + Tevimbra).Not a Keytruda substitute. The 2026 GEA label is a HER2-selected triplet with zanidatamab, not a PD-L1-unselected 1L PD-1 row.Exploits a tight-junction protein that only becomes accessible after malignant transformation, creating tumor selectivity without a novel modality.novel calcineurin inhibitor with predictable pharmacokinetics, increases complete renal response rates and rapidly reduces pr
Positioning
Known limitation
resistance to trastuzumab-based targeted therapy and limited intratumoral antibody penetration continue to restrict clinical outcomesHER2 heterogeneity, PI3K pathway activation, and bypass RTK signaling.Antigen-presentation loss, alternate checkpoints, immunosuppressive TME.CLDN18.2 heterogeneity and loss under treatment pressure.resistance to treatment
Positioning
Development positioning
Challenges the ToGA trastuzumab-chemo backbone in 1L HER2+ GEA; still the only HER2 agent with a dedicated BTC indication.BeOne PD-1 with a China-heavy development history; the US HER2+ GEA triplet is the catalog reason to keep it next to Ziihera, not next to Keytruda's pan-tumor row.Only approved CLDN18.2 agent; CAR-T and ADC competitors are still in trials.
MoA
Mechanism
Trastuzumab binds domain IV of HER2, inhibiting ligand-independent HER2 signaling, mediating ADCC via Fcγ receptors, and inducing internalization/degradation of HER2.Simultaneous binding of two non-overlapping HER2 epitopes crosslinks adjacent receptors, causing receptor clustering, internalization, and degradation alongside ADCC and complement activity.Binds PD-1 on T cells, blocking PD-L1/PD-L2 engagement and restoring antitumor T-cell activity. Fc silencing is intended to avoid macrophage-mediated T-cell clearance.Binds CLDN18.2 on gastric tumor cells and kills them through antibody-dependent cellular cytotoxicity and complement-dependent cytotoxicity.targets type I interferon signaling
MoA
Biomarker
HER2 IHC 3+ or ISH amplified (ASCO/CAP guidelines).GEA: PATHWAY 4B5 IHC and VENTANA Dual ISH. IHC 3+ for chemo doublet; IHC 3+ or IHC 2+/ISH+ when Tevimbra is added. BTC: HER2 IHC 3+.PD-L1 and tumor type on older labels; HER2 IHC/ISH when given with Ziihera.CLDN18.2 IHC with ≥75% of tumor cells at moderate-to-strong membranous staining.biomarker to guide treatment duration in two prospective trials of venetoclax- and sonrotoclax-based triplets
PK/PD
Half-life
4 h~7 days~20 days class range~11 days
PK/PD
Species
Mouse, OS in metastatic BCCynomolgus monkey, Human, OS and PFS in GEA, ORR and DOR in BTCOS/PFS, radiographic responseCynomolgus monkey, Macaque, Mouse
PK/PD
Animal (cat.)
Mouse, In vitroHuman, NHP, In vitroHumanHuman, In vitroMouse, NHP, Monkey
PK/PD
Experiment
PharmacokineticpdpharmacokineticPharmacodynamic
Toxicology
Species
Mouse, RatHumanMouseCynomolgus monkey, Macaque, Mouse
Toxicology
Animal (cat.)
Mouse, NHP, Monkey
Toxicology
Major finding
Cardiotoxicity (LVEF decline, CHF risk especially with anthracyclines); infusion reactions; myelosuppression in combos.Embryo-fetal toxicity warning. Diarrhea, infusion-related reactions, and left ventricular dysfunction require monitoring, as with the HER2 antibody class.Immune-mediated pneumonitis, colitis, hepatitis, endocrinopathies, and infusion-related reactions. Allogeneic transplant complications are labeled.Nausea and vomiting are frequent and often infusion-related, requiring rate adjustment and antiemetics. Hypoalbuminemia, appetite loss, and hypersensitivity also occur.thrombocytopenia or organ dysfunction, were documented
Toxicology
CRS
N/AN/AN/AN/AReported
Clinical
Safety signal
Cardiotoxicity (LVEF decline, CHF risk especially with anthracyclines); infusion reactions; myelosuppression in combos.Embryo-fetal toxicity warning. Diarrhea, infusion-related reactions, and left ventricular dysfunction require monitoring, as with the HER2 antibody class.Immune-mediated pneumonitis, colitis, hepatitis, endocrinopathies, and infusion-related reactions. Allogeneic transplant complications are labeled.Nausea and vomiting are frequent and often infusion-related, requiring rate adjustment and antiemetics. Hypoalbuminemia, appetite loss, and hypersensitivity also occur.thrombocytopenia or organ dysfunction, were documented
Clinical
Selected reported efficacy
ORR 91.2%ORR 52%ORR 87.1%48.1%ORR 100%
Clinical
Reported ORR
91.2%52%87.1%100.0%
Clinical
Reported PFS
~5.5 mo31.5
Clinical
Reported OS
15.54
Clinical
Result source
ClinicalTrials.gov NCT02149524HERIZON-BTC-01 (NCT04466891)ClinicalTrials.gov NCT03209973SPOTLIGHT (NCT03504397) / GLOW (NCT03653507)ClinicalTrials.gov NCT02611323
Clinical
Program phase
APPROVEDAPPROVEDAPPROVEDAPPROVEDPHASE_3
Clinical
Trial activity
No active/completed countsNo active/completed countsNo active/completed countsNo active/completed counts
Clinical
Trial ref
NCT02149524NCT04466891NCT03209973NCT03504397NCT02611323
Preclinical
Animal (cat.)
UnknownMouse, In vitro