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타깃 · 모달리티 · 임상 근거 · 비임상 맥락으로 최대 5개 프로그램을 나란히 비교합니다. · 다음 갱신 D-6 · 마지막 9월 2일

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API CSV32행 · 4개 프로그램

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항목
AntibodyCurated CoreFDAApproved
adalimumab (Humira, Hyrimoz, Amjevita)
AbbVie·TNF-α
574 trials·t½ ~14–19 days (SC)
AntibodyCurated CoreFDAApproved
etanercept (Enbrel, TNFR-Fc)
Amgen / Pfizer·TNF (decoy receptor)
236 trials·t½ 30 h
AntibodyCurated CoreFDAApproved
ustekinumab (Stelara, Wezlana)
Johnson & Johnson·IL-12 / IL-23 (p40)
195 trials·t½ 19 h
AntibodyCurated CoreFDAApproved
guselkumab (Tremfya, CNTO 1959)
Johnson & Johnson·IL-23 p19
87 trials·t½ ~15–18 days
Overview
Program
Humira (adalimumab)Enbrel (etanercept)Stelara (ustekinumab)Tremfya (guselkumab)
Overview
Company
AbbVieAmgen / PfizerJohnson & JohnsonJohnson & Johnson
Overview
Modality
ANTIBODYANTIBODYANTIBODYANTIBODY
Overview
Target
TNF-αTNF (decoy receptor)IL-12 / IL-23 (p40)IL-23 p19
Overview
Indication
Rheumatoid arthritis, psoriasis, Crohn's disease, ulcerative colitis, axial SpARA, PsA, AS, plaque psoriasis, JIAPsoriasis, psoriatic arthritis, Crohn's disease, ulcerative colitisPlaque psoriasis, psoriatic arthritis, ulcerative colitis, Crohn's disease
Overview
Phase
APPROVEDAPPROVEDAPPROVEDAPPROVED
Overview
Status
APPROVEDAPPROVEDAPPROVEDAPPROVED
Overview
Content status
Curated CoreCurated CoreCurated CoreCurated Core
Overview
Data Confidence
Data Confidence · HighData Confidence · HighData Confidence · HighData Confidence · High
Overview
Development Signal
Development Signal · EstablishedDevelopment Signal · EstablishedDevelopment Signal · EstablishedDevelopment Signal · Established
Overview
Approval status
FDA approvedFDA approvedFDA approvedFDA approved
Positioning
Key differentiator
novel object recognition testnovel ADAM17 inhibitor drugs to block soluble TNF-α production in inflammatory diseasesNovel therapeutic avenues, including TYK2 and HDAC inhibitors, as well as nanotechnology-based delivery systems, show encouraLeaving IL-12 intact preserves Th1 antimicrobial immunity while shutting down the Th17 axis, which is why durability is high and infection signals are low.
Positioning
Known limitation
Anti-drug antibodies, TNF-independent inflammation, and immunogenicity lowering trough levels.Anti-drug antibodies (lower vs mAb TNF inhibitors), non-TNF pathways.IL-23 axis escape, anti-drug antibodies, and non-IL-12/23 inflammation.Anti-drug antibodies are rare; non-IL-23-driven fibrostenotic disease does not respond.
Positioning
Development positioning
IL-23 class benchmark alongside Skyrizi, now with the broadest IBD label progression.
MoA
Mechanism
Adalimumab binds specifically to TNF-α and blocks its interaction with p55 and p75 cell-surface TNF receptors, neutralizing TNF-mediated inflammatory cascades (IL-6, adhesion molecules, acute-phase proteins).Etanercept is a dimeric fusion protein of human p75 TNF receptor linked to IgG1 Fc; it binds TNF-α and lymphotoxin-α, preventing receptor activation and downstream NF-κB inflammatory signaling.Ustekinumab binds the p40 subunit shared by IL-12 and IL-23, preventing their interaction with IL-12Rβ1 receptor and downstream Th1/Th17 differentiation and cytokine production.Neutralizes IL-23 by binding p19, blocking IL-23R signaling and downstream Th17/Th22 differentiation, IL-17 production, and tissue inflammation in skin, joints, and gut mucosa.
MoA
Biomarker
CRP, ESR, disease-specific scores (DAS28, PASI, Mayo for IBD).DAS28, PASI, CRP.biomarkers but require external validation in independent cohortsPASI/IGA in psoriasis, ACR20 in PsA, endoscopic improvement and fecal calprotectin in IBD.
PK/PD
Half-life
~14–19 days (SC)30 h19 h~15–18 days
PK/PD
Species
Rat, xenografts, which corroborat, CRP reduction, clinical remission scoresRat, CRP, joint counts, skin scoresMouse, Skin clearance (PASI75/90), endoscopic response in IBDCynomolgus monkey, PASI 90/100, ACR20/50, endoscopic remission, serum IL-17A/IL-22
PK/PD
Animal (cat.)
RatRat, In vitroMouseHuman, NHP
PK/PD
Experiment
pdpharmacokineticPharmacokineticPharmacokinetic
Toxicology
Species
Cynomolgus monkey, Rat, xenografts, which corroboratRatCynomolgus monkey, MouseMouse, Pig
Toxicology
Animal (cat.)
NHP
Toxicology
Major finding
Serious infections (TB reactivation, invasive fungal), malignancy risk signal, injection-site reactions, and rare demyelinating events.Injection-site reactions, infections, rare demyelination and lupus-like syndrome.Infections, reversible posterior leukoencephalopathy syndrome (rare), hypersensitivity.Upper respiratory infection, headache, and injection-site reactions dominate. Serious infection and malignancy signals have stayed low across long-term psoriasis follow-up.
Toxicology
CRS
N/AN/AN/AN/A
Clinical
Safety signal
Serious infections (TB reactivation, invasive fungal), malignancy risk signal, injection-site reactions, and rare demyelinating events.Injection-site reactions, infections, rare demyelination and lupus-like syndrome.Infections, reversible posterior leukoencephalopathy syndrome (rare), hypersensitivity.Upper respiratory infection, headache, and injection-site reactions dominate. Serious infection and malignancy signals have stayed low across long-term psoriasis follow-up.
Clinical
Selected reported efficacy
73.4%PASI75 67%
Clinical
PASI75
PASI 75 response rate at Week 12.67%
Clinical
Result source
ClinicalTrials.gov NCT02405780ClinicalTrials.gov NCT01891864PHOENIX
Clinical
Program phase
APPROVEDAPPROVEDAPPROVEDAPPROVED
Clinical
Trial activity
No active/completed countsNo active/completed countsNo active/completed countsNo active/completed counts
Clinical
Trial ref
NCT02405780NCT01891864PHOENIX
Preclinical
Animal (cat.)
RatMouse