← 홈

프로그램 비교

타깃 · 모달리티 · 임상 근거 · 비임상 맥락으로 최대 5개 프로그램을 나란히 비교합니다. · 다음 갱신 D-6 · 마지막 9월 2일

현재 선택: 2

프로그램 상세에서 관심 등록 후 2개 이상 모으면 여기서 한 번에 비교할 수 있습니다.

API CSV27행 · 2개 프로그램

표가 넓으면 좌우로 스크롤하세요. 핵심 비교 모드에서는 중요 항목만 표시됩니다.

항목
CGTCurated CoreFDAApproved
voretigene neparvovec (Luxturna, AAV2-hRPE65v2, voretigene neparvovec-rzyl)
Spark Therapeutics / Roche·RPE65 (AAV2)
7 trials
CGTCurated CoreFDAApproved
delandistrogene moxeparvovec (Elevidys, SRP-9001, delandistrogene moxeparvovec-rokl)
Sarepta Therapeutics·Micro-dystrophin (AAVrh74)
12 trials
Overview
Program
Luxturna (voretigene neparvovec)Elevidys (delandistrogene moxeparvovec)
Overview
Company
Spark Therapeutics / RocheSarepta Therapeutics
Overview
Modality
CGTCGT
Overview
Target
RPE65 (AAV2)Micro-dystrophin (AAVrh74)
Overview
Indication
Inherited retinal dystrophy due to confirmed biallelic RPE65 mutationsDuchenne muscular dystrophy (DMD) — ambulatory / label-expanded populations
Overview
Phase
APPROVEDAPPROVED
Overview
Status
APPROVEDAPPROVED
Overview
Content status
Curated CoreCurated Core
Overview
Data Confidence
Data Confidence · HighData Confidence · High
Overview
Development Signal
Development Signal · EstablishedDevelopment Signal · Established
Overview
Approval status
FDA approvedFDA approved
Positioning
Key differentiator
Surgical subretinal delivery; landmark ophthalmology gene therapyMuscle-directed AAV micro-dystrophin vs exon-skipping ASOs
Positioning
Known limitation
bypasses defective phototransduction pathways, showing promise for patients regardless of their genetic profileAcute liver injury, myocarditis, immune myositis
Positioning
Development positioning
Only approved RPE65 gene therapyFirst approved AAV gene therapy for DMD in the US
Technology
Vector
AAV2AAVrh74
MoA
Mechanism
Voretigene neparvovec uses AAV2 to deliver functional RPE65 to retinal pigment epithelium, restoring the visual cycle and improving light sensitivity in RPE65-mutant IRD.Delandistrogene moxeparvovec (AAVrh74) delivers micro-dystrophin transgene to skeletal/cardiac muscle in Duchenne muscular dystrophy ambulatory patients.
MoA
Biomarker
MLMT, FST, BCVA.Micro-dystrophin expression, NSAA motor function.
PK/PD
Species
NHP, Despite these findings, and no systemic toxicity was identified.NHP, Micro-dystrophin Western blot, functional motor scores
PK/PD
Animal (cat.)
NHPHuman, NHP
PK/PD
Experiment
biodistributionpd
Toxicology
Species
NHP, Dog, Despite these findings, and no systemic toxicity was identified.NHP
Toxicology
Animal (cat.)
NHP, Dog
Toxicology
Major finding
Procedure-related ocular AEs; inflammation manageable with steroidsHepatotoxicity; immune-mediated myositis/myocarditis signals
Clinical
Safety signal
Procedure-related ocular AEs; inflammation manageable with steroidsHepatotoxicity; immune-mediated myositis/myocarditis signals
Clinical
Program phase
APPROVEDAPPROVED
Clinical
Trial activity
4 recruiting · 3 completed4 recruiting · 6 completed
Preclinical
Animal (cat.)
NHP