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API CSV33행 · 2개 프로그램

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항목
AntibodyCurated CoreFDAApproved
trastuzumab (Herceptin, Ogivri, Herzuma)
Roche / Genentech·HER2
1100 trials·t½ 4 h
AntibodyCurated CoreFDAApproved
zanidatamab (Ziihera, ZW25, zanidatamab-hrii)
Jazz Pharmaceuticals / Zymeworks·HER2 (biparatopic)
35 trials·t½ ~7 days
Overview
Program
Herceptin (trastuzumab)Ziihera (zanidatamab)
Overview
Company
Roche / GenentechJazz Pharmaceuticals / Zymeworks
Overview
Modality
ANTIBODYANTIBODY
Overview
Target
HER2HER2 (biparatopic)
Overview
Indication
HER2+ breast cancer, HER2+ gastric/GEJ adenocarcinomaFirst-line HER2-positive gastric, GEJ, or esophageal adenocarcinoma; previously treated HER2-positive biliary tract cancer
Overview
Phase
APPROVEDAPPROVED
Overview
Status
APPROVEDAPPROVED
Overview
Content status
Curated CoreCurated Core
Overview
Data Confidence
Data Confidence · HighData Confidence · High
Overview
Development Signal
Development Signal · EstablishedDevelopment Signal · Established
Overview
Approval status
FDA approvedFDA approved
Positioning
Key differentiator
novel bifunctional chelator (2E-C-NETA) for applications in 177 Lu-based TRTDual-epitope binding drives receptor clustering, faster internalization, and stronger signaling blockade than either monoclonal alone. In GEA the label splits IHC 3+ (chemo doublet) from IHC 3+ or 2+/ISH+ (chemo + Tevimbra).
Positioning
Known limitation
resistance to trastuzumab-based targeted therapy and limited intratumoral antibody penetration continue to restrict clinical outcomesHER2 heterogeneity, PI3K pathway activation, and bypass RTK signaling.
Positioning
Development positioning
Challenges the ToGA trastuzumab-chemo backbone in 1L HER2+ GEA; still the only HER2 agent with a dedicated BTC indication.
MoA
Mechanism
Trastuzumab binds domain IV of HER2, inhibiting ligand-independent HER2 signaling, mediating ADCC via Fcγ receptors, and inducing internalization/degradation of HER2.Simultaneous binding of two non-overlapping HER2 epitopes crosslinks adjacent receptors, causing receptor clustering, internalization, and degradation alongside ADCC and complement activity.
MoA
Biomarker
HER2 IHC 3+ or ISH amplified (ASCO/CAP guidelines).GEA: PATHWAY 4B5 IHC and VENTANA Dual ISH. IHC 3+ for chemo doublet; IHC 3+ or IHC 2+/ISH+ when Tevimbra is added. BTC: HER2 IHC 3+.
PK/PD
Half-life
4 h~7 days
PK/PD
Species
Mouse, OS in metastatic BCCynomolgus monkey, Human, OS and PFS in GEA, ORR and DOR in BTC
PK/PD
Animal (cat.)
Mouse, In vitroHuman, NHP, In vitro
PK/PD
Experiment
Pharmacokineticpd
Toxicology
Species
Mouse, RatHuman
Toxicology
Major finding
Cardiotoxicity (LVEF decline, CHF risk especially with anthracyclines); infusion reactions; myelosuppression in combos.Embryo-fetal toxicity warning. Diarrhea, infusion-related reactions, and left ventricular dysfunction require monitoring, as with the HER2 antibody class.
Toxicology
CRS
N/AN/A
Clinical
Safety signal
Cardiotoxicity (LVEF decline, CHF risk especially with anthracyclines); infusion reactions; myelosuppression in combos.Embryo-fetal toxicity warning. Diarrhea, infusion-related reactions, and left ventricular dysfunction require monitoring, as with the HER2 antibody class.
Clinical
Selected reported efficacy
ORR 91.2%ORR 52%
Clinical
Reported ORR
91.2%52%
Clinical
Reported PFS
~5.5 mo
Clinical
Reported OS
15.54
Clinical
Result source
ClinicalTrials.gov NCT02149524HERIZON-BTC-01 (NCT04466891)
Clinical
Program phase
APPROVEDAPPROVED
Clinical
Trial activity
No active/completed countsNo active/completed counts
Clinical
Trial ref
NCT02149524NCT04466891
Preclinical
Animal (cat.)
Unknown